Color Atlas of Fetal and Neonatal Histology by Linda M. Ernst MD, MHS (auth.), Linda M. Ernst, Eduardo D.

By Linda M. Ernst MD, MHS (auth.), Linda M. Ernst, Eduardo D. Ruchelli, Dale S. Huff (eds.)

This e-book serves as a accomplished atlas of the developmental histology of the foremost organs from 15 weeks gestation to the neonatal interval. each one part contains textual content on uncomplicated embryologic tactics that impression the improvement of every organ and spotlight significant histologic good points that correspond with definite developmental classes. moreover, there are lots of colour photomicrographs at key developmental phases to help the reader in opting for applicable histologic adjustments at each one developmental degree. This e-book could be of significant price to scholars of embryology, pathology citizens and fellows, and attending pathologists who practice fetal autopsies.

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The formation of a single-layered capillary network in concert with the differentiation of airspace epithelium to flattened pneumocytes provides for the maturing capillary–airspace interface necessary for eventual postnatal respiration [1,6,10]. Figure 2-31. Early saccular stage I: lung at 28 weeks ­gestation. The early saccular stage is characterized by more complex airspaces with decreasing amounts of interstitial mesenchyme and an expanding capillary network. ) Figure 2-32. Early saccular stage II: lung at 28 weeks ­gestation.

DePaepe ME: Lung growth and development. In Thurlbeck’s Pathology of the Lung, edn 3. Edited by Churg AM, Myers JL, Tazelaar HD, Wright JL. New York: Thieme; 2005:39–71. 2. Colby TV, Leslie KO, Yousem SA: Lungs. In Histology for Pathologists, edn 3. Edited by Mills SE. Philadelphia: Lippincott Williams & Wilkins; 2007:473–483. 3. Langston C: Prenatal lung growth and pulmonary hypoplasia. In Pediatric Pulmonary Disease. Edited by Stocker JT. New York: Hemisphere; 1989:1–13. 4. Kimura J, Deutsch GH: Key mechanisms of early lung development.

3-7 and 3-8). Esophageal cardiac glands, which are similar to gastric cardiac or pyloric glands, are mucous-producing glands in the lamina propria connected to the lumen by a duct that passes through the epithelium. Papillae of the lamina propria, which are finger-like processes of lamina propria that penetrate up into the epithelium during postnatal life and are important in the evaluation of esophagitis, are not present during fetal life (see Fig. 3-7). The esophageal muscularis mucosae rapidly increases in thickness and becomes the thickest of all the segments of the gastrointestinal tract.

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